TAT‐dextran–mediated mitochondrial transfer enhances recovery from models of reperfusion injury in cultured cardiomyocytes
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DOI: 10.1111/jcmm.15120
Abstract: Acute myocardial infarction is a leading cause of death among single organ diseases. Despite successful reperfusion therapy, ischaemia reperfusion injury (IRI) can induce oxidative stress (OS), cardiomyocyte apoptosis, autophagy and release of inflammatory cytokines, resulting… read more here.
Keywords: reperfusion injury; mitochondrial transfer; tat dextran; transfer ... See more keywords