Long-Term Latency of Highly Mutated Cells in Normal Mouse Skin Is Reversed by Exposure to Tumor Promoters and Chronic Tissue Damage
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DOI: 10.1158/2159-8290.cd-24-1379
Abstract: Tumor initiation, via either chemical mutagens or genetic models of Ras activation, produces long-lived, but latent, mutated cells that require chronic tumor promoter exposure to form cancers. read more here.
Keywords: long term; highly mutated; term latency; tumor ... See more keywords