G2A as a Threshold Regulator of Inflammatory Hyperalgesia Modulates Chronic Hyperalgesia
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DOI: 10.1007/s12031-017-1000-3
Abstract: Tissue injury, pathogen infection, and diseases are often accompanied by inflammation to release mediators that sensitize nociceptors and further recruit immune cells, which can lead to chronic hyperalgesia and inflammation. Tissue acidosis, occurring at the… read more here.
Keywords: chronic hyperalgesia; inflammatory hyperalgesia; hyperalgesia; threshold regulator ... See more keywords