The β3-subunit modulates the effect of venom peptides ProTx-II and OD1 on NaV 1.7 gating.
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DOI: 10.1002/jcp.31018
Abstract: The voltage-gated sodium channel NaV 1.7 is involved in various pain phenotypes and is physiologically regulated by the NaV -β3-subunit. Venom toxins ProTx-II and OD1 modulate NaV 1.7 channel function and may be useful as… read more here.
Keywords: venom; treated nav; od1 nav; od1 treated ... See more keywords