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Published in 2018 at "Genome Medicine"
DOI: 10.1186/s13073-018-0530-9
Abstract: BackgroundCerebral amyloidosis, neuroinflammation, and tauopathy are key features of Alzheimer’s disease (AD), but interactions among these features remain poorly understood. Our previous multiscale molecular network models of AD revealed TYROBP as a key driver of…
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Keywords:
pathological interactions;
molecular pathological;
biology;
tau trem2 ... See more keywords