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Published in 2017 at "Journal of Alzheimer's Disease"
DOI: 10.3233/jad-170223
Abstract: Background: Identifying physiologically relevant binding partners of amyloid-β (Aβ) that modulate in vivo fibril formation may yield new insights into Alzheimer’s disease (AD) etiology. Human cathelicidin peptide, LL-37, is an innate immune effector and modulator,…
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Keywords:
two peptides;
evidence;
evidence human;
fibril formation ... See more keywords