AMPK phosphorylation of FNIP1 (S220) controls mitochondrial function and muscle fuel utilization during exercise
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DOI: 10.1126/sciadv.adj2752
Abstract: Exercise-induced activation of adenosine monophosphate–activated protein kinase (AMPK) and substrate phosphorylation modulate the metabolic capacity of mitochondria in skeletal muscle. However, the key effector(s) of AMPK and the regulatory mechanisms remain unclear. Here, we showed… read more here.
Keywords: ampk; phosphorylation; muscle; utilization exercise ... See more keywords