The function of RAD52 N-terminal domain is essential for viability of BRCA-deficient cells
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DOI: 10.1093/nar/gkaa1145
Abstract: Abstract RAD52 is a member of the homologous recombination pathway that is important for survival of BRCA-deficient cells. Inhibition of RAD52 leads to lethality in BRCA-deficient cells. However, the exact mechanism of how RAD52 contributes… read more here.
Keywords: deficient cells; brca deficient; viability brca; deficient ... See more keywords